Insulin Resistance is Caused by Infection
Insulin resistance is involved in most chronic diseases. It is not just diabetes or so called metabolic syndrome.
Psoriasis and other “autoimmune” considered diseases are accompanied by insulin resistance, elevated fasting blood glucose sugar and hypoglycemic episodes.
Hormonal imbalances are actually often caused by gut imbalances (SIBO and fungi with other parasites) which lead to inflammation.
Insulin is a hormone which is essential for the body to regulate the blood sugar and maintaining the homeostasis. It is a powerful hormone which directly or indirectly affects most of the other hormones and their effects on the body. When there is an inflammation in the intestines (mostly small intestine) your hormonal system can’t be OK.
Insulin resistance often leads to hypoglycemia which in turn causes the adrenaline production. That is why we get angry and nervous when we are hungry. That’s why there is so much behavioral problems in kids who eat a lot of sweets.
It is not about the table sugar as molecule (glucose+fructose), it is about the hypoglycemia which is often induced by rapidly absorbed sugar (drinking a cup of soda will get you about 20 grams of sugar) which instantly increases the insulin release.
However, at least in my opinion drinking sweet sodas or eating fruit is not the worst you can do for insulin sensitivity.
The worst is infection
There are millions of people who don’t realize that their dental infections and root canal teeth are making them sick. So many people cured their diabetes just by having their root canal teeth removed.
The results were usually amazingly fast – like 2 or 3 days after an extraction of teeth their blood sugar dropped significantly. Not due to changes in their diet, it’s just because they removed the infected part of their body – removed the source of toxins and reduced the inflammation pathways.
Insulin resistance caused by infection
It is scientifically documented that acute infections are accompanied by insulin resistance, worsening of blood sugar control in diabetics as well as decreased glucose tolerance in non-diabetics. The authors of study from 1984 concluded that binding of insulin to cells was not affected by acute infection. Therefore the insulin resistance in infection is mediated at the post-receptor level. That means that there is some problem in the inside of the cells which leads to symptoms of insulin resistance.
The shocking is the data from scientific paper published in JAMA in 1943. The authors state:
“Infection is probably the most common of the several causes for insulin resistance in diabetes mellitus in man, but the mechanism of this phenomenon is not understood. There are several theories which have been advocated to explain this resistance: (1) decrease of endogenous insulin secretion by the pancreas, (2) decrease of effectiveness of endogenous and exogenous insulin, (3) increase of metabolic rate, (4) greater glycogenolysis by the liver, (5) increase of epinephrine secretion by the adrenal, (6) decrease of insulin kinase secreted by the liver, (7) increase of adrenal cortex secretion, (8) increase of secretion by the anterior lobe of the pituitary gland, (9) increase of secretion by the thyroid gland, (10) decrease in ability of the skeletal muscles to form glycogen and (11) increase of inhibitory action on insulin of the blood plasma.”
Would you believe that? So much truth in one of the most popular and respected medical journals and nobody cares?
The authors concluded that infection is probably the most common cause of insulin resistance!
Now we are 72 years later and the best diabetes treatment medicine offers is pumping the patients with insulin injections?
Type 2 diabetics produce enough of insulin, the problem is that cells do not respond to it as they should. The cells are hypo-responsive to insulin