Alcohol Is Not As Bad For Psoriasis As You Think

Alcohol is considered to be a risk factor for psoriasis but most people do not know why and how it may induce or worsen psoriasis.

Now let me explain that…

Alcohol directly does not cause psoriasis but at the same time it may significantly worsen it. Endotoxemia is perhaps the worst acute problem caused by alcohol consumption.

The biggest part in psoriasis development in most people is probably the liver function.

Liver is a major “biochemical factory” so when you drink any alcohol and your psoriasis worsen, become more red or itchy it is the most likely a fault of the liver.

As I said above “alcohol is considered to be a risk factor for psoriasis”. I said “considered” because the science claims that there’s not enough evidence whether alcohol consumption is a risk factor for psoriasis. [1]

I have to agree with that.

Why?

You know, maybe those who drink excessive amounts of alcohol do that because they feel anxious about their psoriasis or cause of psoriasis (which is a nervous system disorder) shares some underlying processes with anxiety disorders which many people cope with by drinking alcohol.

In other words, if somebody started drinking alcohol after he developed psoriasis then alcoholism should not be considered as a cause of psoriasis rather then the result of psoriasis-induced low self-esteem anxiety.

However, generally speaking, I can tell you that everything what places a burden on the liver is indeed a risk factor for a disease. Not only for psoriasis but actually for any chronic degenerative disease.

Is there a connection between alcohol and psoriasis?

Yes, study on 100 patients with chronic plaque psoriasis concluded that in male patients heavy drinking at level which is detrimental to health was found more commonly in those with severe psoriasis. However, in women excess alcohol intake does not correlated as much with the severity of psoriasis as in male patients. [2]

Another study examined 1987 patients (due to whatever the diagnosis was present) for the presence or absence of psoriasis and alcohol consumption. [13]

Image Source: JEAN-CLAUDE CHAPUT, MD, hepatologist; THIERRY POYNARD, MD, PHD, hepatologist; SYLVIE NAVEAU, MD, hepatologist; DOMINIQUE PENSO, MD, dermatologist; OLIVIER DURRMEYER, MD, graduate; DENIS SUPLISSON, MD, graduate. Psoriasis, alcohol, and liver disease. BRITISH MEDICAL JOURNAL VOLUME 291, 6 JULY 1985 [13]

The incidence of psoriasis in patients who regularly consumed alcohol was 5,3% compared to non-drinkers 0,7%.

 


Epidemiological evidence suggests that patients with moderate to severe psoriasis have an increased incidence of alcohol-related diseases and mortality. This appears to be unique to psoriasis compared with other autoimmune diseases.
” [3]

The excessive consumption of alcohol may significantly contribute to inflammation, cardiovascular disease and depression. [3]

Endotoxins, alcohol and psoriasis

Endotoxins are undoubtedly a big cause of inflammation and chronic inflammatory diseases like diabetes, chronic hearth failure, periodontal diseases and others. [8, 9, 10]

That is why we should always look for a possible cause or source of endotoxins like small intestine inflammation or dental infection in every developed chronic degenerative disease.

Alcohol significantly affects the liver and intestinal health which may affect the levels of circulating endotoxins in the whole blood.

Higher levels of circulating endotoxins are observed in alcohol-abusing patients. [4]


Patients with minor (A1; n=27), more pronounced (A2; n=13), cirrhotic alcoholic liver disease (A3; n=18), and non-alcoholic cirrhosis (NC; n=6), and 15 healthy control persons (HC) were included in the study.

All groups of alcohol abusers had significantly elevated endotoxin plasma levels with a considerable portion of ‘bound’ endotoxin. Conversely, the endotoxin binding capacity was markedly diminished, mainly in patients with more advanced liver disease (A1: 85.8% of the control value [non-significant vs. controls]; A2: 25.4% [p<0.05]; A3: 43.6% [p<0.02], NC: 43.2%).

The endotoxin-binding capacity is diminished

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